2015年12月8日星期二

Treatment for IgA nephropathy with renal function 38%

IgA nephropathy is an autoimmune disease with deposits of immunoglobulin A (IgA) in area mesangial.Sin aggressive treatment, renal function decline progresivamente.¿Cuál is the treatment for IgA nephropathy with 38% kidney function?

IgA nephropathy with renal function 38%

Immune system is a powerful protection cuerpo.Cuando pathological substances invade the body, the immune system will be activated to produce antibodies to derrotarlos.Después harmful substances are removed, the immune system will stop working.

In autoimmune disorder, however, the immune system does not work and additional antibodies, particularly IgA are producidos.Con bloodstream, IgA enter glomeruli and then stay in area mesangial.Los immune deposits can trigger inflammation in riñones.Si not aggressive treatment is applied , inflammation worsen renal function, resulting in chronic kidney disease (CKD).

Based on renal function, CKD can be divided into five stages. A person with IgA nephropathy in kidney function is 38% Stage 3 CKD.En this stage, as the kidneys are only moderately damaged, patients still have a high probability to reverse kidney damage.

Treatment for IgA nephropathy with renal function 38%

Conservative treatment of IgA nephropathy only inmunosupresores.Estos agents include hormones and drugs can suppress inflammation decrease renal function in certain medida.Sin however, have no point in improving renal function.

To reverse kidney damage in IgA nephropathy, immunotherapy and hot compress therapy will be applied throughout the treatment process.

Immunotherapy can correct the immune dysfunction and help the body recover normal.El immune system immune system will protect the body other than attack kidney tissues.

As for hot compress therapy, you can activate the self-healing and self-regenerating kidney tissue renales.Cuando structure is restored, renal function will be improved notablemente.Si so, IgA nephropathy with renal function 38% will be invested .


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