With kidney involvement, there may be a loss of small amounts of blood and protein in the urine, but this usually goes unnoticed. Frequent relapse is a significant risk factor in aggravating renal function in purpura nephritis. Every relapse can trigger the inflammation in kidneys, which can worsen renal function. Therefore, even if your ill keeps stable, you still should go to see a doctor and do regular monitoring.
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If women have had Henoch-Schonlein purpura in the early years of their life, they are at higher risk of developing high blood pressure during pregnancy than the general population. Therefore, if you have a history of Henoch-Schonlein, you should report it to your doctor and are monitored closely during pregnancy.
The etiology of Pediatric Henoch-Schoenlein Purpura remains unknown. However, IgA clearly plays a critical role in the immunopathogenesis of Henoch-Schoenlein purpura, as evidenced by increased serum IgA concentrations, IgA-containing circulating immune complexes, and IgA deposition in vessel walls and renal mesangium. Henoch-Schoenlein purpura is almost exclusively associated with abnormalities involving IgA1, rather than IgA2.
IgA is the short name of immunoglobulin A, which is released by immune system. When immune system cannot work well, the IgA 1 or IgA 2 will be extra, causing problems, such as Pediatric Henoch-Schonlein Purpura or IgA Nephropathy. Thereby, many experts believe that Pediatric Henoch-Schonlein Purpura is caused by diseased immune system.
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